Searching journal content for articles similar to Zhang et al. 23 (6): 917.

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  1. Research: A dynamic and integrated epigenetic program at distal regions orchestrates transcriptional responses to VEGFA
    • Shiyan Wang,
    • Jiahuan Chen,
    • Sara P. Garcia,
    • Xiaodong Liang,
    • Fang Zhang,
    • Pengyi Yan,
    • Huijing Yu,
    • Weiting Wei,
    • Zixuan Li,
    • Jingfang Wang,
    • Huangying Le,
    • Zeguang Han,
    • Xusheng Luo,
    • Daniel S. Day,
    • Sean M. Stevens,
    • Yan Zhang,
    • Peter J. Park,
    • Zhi-jie Liu,
    • Kun Sun,
    • Guo-cheng Yuan,
    • William T. Pu,
    • and Bing Zhang
    Genome Res. February 2019 29: 193-207; Published in Advance January 22, 2019, doi:10.1101/gr.239053.118
    ...,B; Supplemental Fig. S4A–C). Forty-eight percent of SEs were present at all four time points, while the remaining 52% were dynamic under VEGFA stimulation, changing at least at one time point (Fig. 4C), and the average H3K27ac signal at all SEs (Fig. 4D and SE targeted genes; Supplemental Fig. S4D) changed...
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  2. Research: TP53 engagement with the genome occurs in distinct local chromatin environments via pioneer factor activity
    • Morgan A. Sammons,
    • Jiajun Zhu,
    • Adam M. Drake,
    • and Shelley L. Berger
    Genome Res. February 2015 25: 179-188; Published in Advance November 12, 2014, doi:10.1101/gr.181883.114
    ...reported. Chromatin modification dynamics at TSS have been linked to changes in TP53 activity. For example, coordinate regulation of H3K4me3 (MLL complexes) and H4K16ac (KAT8) at TSS activates TP53-dependent transcription (Dou et al. 2005). Similarly, coordination of H3K4me3 and H3K27ac by CREBBP/EP300...
  3. Research: Vascular histone deacetylation by pharmacological HDAC inhibition
    • Haloom Rafehi,
    • Aneta Balcerczyk,
    • Sebastian Lunke,
    • Antony Kaspi,
    • Mark Ziemann,
    • Harikrishnan KN,
    • Jun Okabe,
    • Ishant Khurana,
    • Jenny Ooi,
    • Abdul Waheed Khan,
    • Xiao-Jun Du,
    • Lisa Chang,
    • Izhak Haviv,
    • Samuel T. Keating,
    • Tom C. Karagiannis,
    • and Assam El-Osta
    Genome Res. August 2014 24: 1271-1284; Published in Advance April 14, 2014, doi:10.1101/gr.168781.113
    ...G methylation at target genes responsive to HDAC inhibition is poorly understood, and one explanation is that sites of histone acetylation are protected from HDAC inhibition at HCPs. Recent studies have shown that H3K4me3 and H3K79me1 are more predictive for gene activity at LCPs, whereas H3K27ac and H4K20me1...
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