Searching journal content for articles similar to Welle et al. 9 (5): 506.

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  1. ...for uncovering the molecular basis of cancer. Nat. Genet. 15: 415-416. Velculescu, V.E., L. Zhang, B. Vogelstein, and K.W. Kinzler. 1995. Serial analysis of gene expression. Science 270: 484–487. Welle, S., K. Bhatt, and C.A. Thornton. 1999. Inventory of high-abundance mRNAs in skeletal muscle of normal men...
  2. ....W. ( 1995 ) Serial analysis of gene expression. Science 270 : 484 – 487 . ↵ Welle S. , Bhatt K. , Thornton C.A. ( 1999 ) Inventory of high-abundance mRNAs in skeletal muscle of normal men. Genome Res. 9 : 506 – 513 . ↵ Wickens M. , Stephenson P. ( 1984 ) Role of the conserved AAUAAA sequence: Four AAUAAA...
  3. ...of mitochondrial DNA (mtDNA) copies in a cell. Intrinsically, modification of the number of mtDNA copies in a cell could allow concomitant variation in cellular concentration of mitochondrial mRNAs ( Williams 1986 ; Kaufmann et al. 1996 ). However, this mechanism has not been supported by other data that indicate...
  4. ...of the human . Science 291 : 1304 – 1351 . ↵ Welle S. , Bhatt K. , Thornton C.A. ( 1999 ) Inventory of high-abundance mRNAs in skeletal muscle of normal men. Genome Res. 9 : 506 – 513 . Pattern of Organization of Human Mitochondrial Pseudogenes in the Nuclear Genome Markus Woischnik and Carlos T. Moraes 1...
  5. ...levels. Moreover, ESTs may derive from unspliced immature mRNAs, alternative splicing and polyadenylation sites, cloning artifacts (chimerisms), and mitochondrial (mt) transcripts. Depending on tissue source and RNA extraction/fractioning methods, a remarkably high fraction of ESTs from a given c...
  6. ...may cause underestimation of the actual frequency for some abundant mRNA species in highly specialized tissues, as was recently demonstrated in a SAGE study of human skeletal muscle ( Welle et al. 1999 ). However, it remains to be seen whether a combined GST long SAGE–based approach that relies...
  7. ....A. , Marchuk D.A. , Warman M.L. , Cantley L.C. , et al. ( 1996 ) Vascular dysmorphogenesis caused by an activating mutation in the receptor tyrosine kinase TIE2. Cell 87 : 1181 – 1190 . ↵ Welle S. , Bhatt K. , Thornton C.A. ( 1999 ) Inventory of high-abundance mRNAs in skeletal muscle of normal men. Genome Res...
  8. ...in human fibroblasts. Am. J. Hum. Genet. 31 : 478 – 488 . ↵ Welle S. , Bhatt K. , Thornton C.A. ( 1999 ) Inventory of high-abundance mRNAs in skeletal muscle of normal men. Genome Res. 9 : 506 – 513 . ↵ Whatley S.A. , Hall C. , Davison A.N. , Lim L. ( 1984 ) Alterations in the relative amounts of specific...
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