Searching journal content for articles similar to Fei et al. 25 (9): 1325.

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  1. Review: The superpowers of imprinting control regions
    • Bertille Montibus,
    • Franck Court,
    • and Philippe Arnaud
    Genome Res. January 2026 36: 1-19; Published in Advance December 10, 2025, doi:10.1101/gr.281215.125
    ...a hexanucleotide sequence present in multiple copies within most ICRs and binds to this sequence only in its methylated form. ZFP57 then serves as a platform for assembling the KRAB repressor complex, which includes TRIM28 and SETDB1, a histone methyltransferase that deposits H3K9me3. These factors, in turn...
  2. Research: Multi-omics analyses demonstrate a critical role for EHMT1 methyltransferase in transcriptional repression during oogenesis
    • Hannah Demond,
    • Courtney W. Hanna,
    • Juan Castillo-Fernandez,
    • Fátima Santos,
    • Evangelia K. Papachristou,
    • Anne Segonds-Pichon,
    • Kamal Kishore,
    • Simon Andrews,
    • Clive S. D'Santos,
    • and Gavin Kelsey
    Genome Res. January 2023 33: 18-31; Published in Advance January 23, 2023, doi:10.1101/gr.277046.122
    ...the repressive chromatin landscape in a subset of regions in the oocyte. This could reflect its action in depositing H3K9me1/2, its activity as a transcriptional repressor, or its modulation of H3K27me2/3 levels, as EHMT1/2 have been reported to interact with and contribute to recruitment of PRC2 in mouse ESCs...
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  3. Research: Characterization of the distribution and dynamics of chromatin states in the C. elegans germline reveals substantial H3K4me3 remodeling during oogenesis
    • Mariateresa Mazzetto,
    • Lauren E. Gonzalez,
    • Nancy Sanchez,
    • and Valerie Reinke
    Genome Res. January 2024 34: 57-69; Published in Advance December 26, 2023, doi:10.1101/gr.278247.123
    ...comprise undifferentiated proliferating and early meiotic germ cells (Supplemental Fig. S1A; Han et al. 2019). Data sets include profiles of five well-characterized histone marks (H3K27me3, H3K9me3, H3K36me3, H3K4me3, and H3K27ac), transcript abundance, and chromatin accessibility data (Methods...
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  4. Research: PML protein organizes heterochromatin domains where it regulates histone H3.3 deposition by ATRX/DAXX
    • Erwan Delbarre,
    • Kristina Ivanauskiene,
    • Jane Spirkoski,
    • Akshay Shah,
    • Kristin Vekterud,
    • Jan Øivind Moskaug,
    • Stig Ove Bøe,
    • Lee H. Wong,
    • Thomas Küntziger,
    • and Philippe Collas
    Genome Res. June 2017 27: 913-921; Published in Advance March 24, 2017, doi:10.1101/gr.215830.116
    ...and Pandolfi 2007), linking PML to gene activity. In contrast, PML bodies can also associate with transcriptional repressors such as histone deacetylases (Khan et al. 2001), CBX3 (Seeler et al. 1998; Ivanauskiene et al. 2014), and the H3K9 methyltransferase SETDB1 (Cho et al. 2011). This implicates PML...
  5. Research: A heterozygous IDH1R132H/WT mutation induces genome-wide alterations in DNA methylation
    • Christopher G. Duncan,
    • Benjamin G. Barwick,
    • Genglin Jin,
    • Carlo Rago,
    • Priya Kapoor-Vazirani,
    • Doris R. Powell,
    • Jen-Tsan Chi,
    • Darell D. Bigner,
    • Paula M. Vertino,
    • and Hai Yan
    Genome Res. December 2012 22: 2339-2355; Published in Advance August 16, 2012, doi:10.1101/gr.132738.111
    ...globally increased, consistent with broad inhibition of histone demethylation, hypermethylation at H3K9 in particular accompanied locus-specific DNA hypermethylation at several genes down-regulated in IDH1 R132H/WT knock-in cells. These data provide insight on epigenetic alterations induced by IDH1...
  6. Research: TRIM28 repression of retrotransposon-based enhancers is necessary to preserve transcriptional dynamics in embryonic stem cells
    • Helen M. Rowe,
    • Adamandia Kapopoulou,
    • Andrea Corsinotti,
    • Liana Fasching,
    • Todd S. Macfarlan,
    • Yara Tarabay,
    • Stéphane Viville,
    • Johan Jakobsson,
    • Samuel L. Pfaff,
    • and Didier Trono
    Genome Res. March 2013 23: 452-461; Published in Advance December 10, 2012, doi:10.1101/gr.147678.112
    ...;Ooi et al. 2007; Weber et al. 2007; Stadler et al. 2011), and SETDB1 has previously been shown to maintain H3K9 trimethylation and, secondarily, the Suv420H1/2-mediated mark H4K20me3 at ERVs (Matsui et al. 2010). Inactivation of this machinery leads not only to the loss of silent histone marks...
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