Mechanisms, contributing factors, predictions, and evidence for biased genome-wide TE distributions according to chromosome type: sex chromosomes (X, Y) versus autosomes (A)
[i] aGenome landscape features used as predictors in models of TE densities (for details, see Supplemental Table S2 and Methods).
[ii] bGC content, L1 target site density, Telomere-containing hexamer frequency, Replication timing, CpG island density, CpG content, Nucleosome-free region density, and Germline-expressed region density are variables used to model local TE integration preferences (for details, see Supplemental Table S2).
[iii] cGenome landscape features significant to predicting variability in TE densities vary for individual subfamilies (for model-specific details, see Fig. 1 and Supplemental Table S4).
[iv] NA, Not available.